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The role of GLP-1 in the nervous system remains inconclusive, yet indicates its potential for alleviating neuroinflammation, safeguarding neurons and glial cells from oxidative damage, and optimizing neurotransmitter equilibrium [83]
A aggregation and tau hyperphosphorylation can disrupt the calcium balance in neurons, leading to mitochondrial calcium overload, increased reactive oxygen species production, reduced ATP generation, and ultimately, neuronal apoptosis or necrosis [76, 77]
Nausea, vomiting, and diarrhea occurred commonly during dose escalation phases
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