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Experimental investigations commonly focus on its interaction with glutathione peroxidases, glutathione S-transferases, and redox-sensitive signalling proteins

Lastly, the dose is a problem.

doi:10.1016/j.ophtha.2024.03.004 5

This HO-1-dependent cytoprotective mechanism may explain much of BPC-157s observed ability to protect cells against ischaemia-reperfusion injury and oxidative damage across multiple tissue types simultaneously