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For example, C16-carnitine (10 M) may impair mitochondrial function in rat ventricular myocytes by accelerating ROS production and increasing the probability of mitochondrial permeability transition pore (mPTP) opening, whereas low concentrations (1 and 5 M) of C16-carnitine lightly hyperpolarize the m but do not open the mPTP [293]
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Carnitine has been demonstrated to exert beneficial effects on the structure and development of white matter in the brain [94, 95]
Cross-talk between hepatic stellate cells and T lymphocytes in liver fibrosis
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