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That added component is what shifts the blend more clearly toward inflammation-related and barrier-oriented research logic, rather than a repair-only emphasis

We conclude that the main function of endogenous GSH is not to limit inflammation but to fine-tune the innate immune response to infection

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Another transporter, CACT (carnitine-acylcarnitine translocase) is located in the inner mitochondrial membrane and contributes to the exchange of acylcarnitine and longer acylcarnitines for carnitine to ensure acetylcarnitine transfer across the inner mitochondrial membrane [90]

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