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Insulin-like growth factors I and II peptide and messenger RNA levels in macrosomic infants of diabetic pregnancies
Piling on more unbuffered acid onto this structural barrier mismatch is entirely counterproductive
Although pre-clinical and clinical confirmation is required, these studies suggest that PTC may in part be driven by UCA1 acting as a ceRNA to suppress miR-204-5p and stimulate oncogenic IGFBP-5 [70]
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