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These modifications can include the addition of lipid groups (lipoproteins) to anchor proteins, phosphorylation to regulate protein activity, methylation for chemotaxis, and N-acylation to integrate proteins into the lipid bilayer

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In keratinocytes, GHK-Cu has been shown to downregulate inflammatory cytokine production in response to challenge including IL-1, TNF-, and IL-6, the inflammatory mediators most associated with the chronic scalp inflammation seen in androgenetic alopecia.4 In fibroblasts, GHK-Cu stimulates collagen and elastin synthesis, supporting the structural matrix that follicles depend on for anchoring and nutrient supply

Supplementary Data 2 (download XLSX DESeq data from RNA analysis

Br J Diabetes Vasc Dis 2004;4:401406 Qureshi SA, Ainsworth A, Winocour PH