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The expression of GST A3-3 in steroidogenic tissues suggests that the enzyme has evolved to support the activity of 3-hydroxysteroid dehydrogenase, which catalyzes the formation of 5-androsten-3,17-dione and 5-pregnen-3,20-dione that are substrates for the double-bond isomerization catalyzed by GST A3-3
The precise network processes of crosstalk governing OA ferroptosis and other types of autophagy need comprehensive investigation to pinpoint novel treatment targets
Dipeptidyl peptidase-4 inhibitors and gallbladder or biliary disease in type 2 diabetes: systematic review and pairwise and network meta-analysis of randomised controlled trials
Oxidative stress and psychological disorders
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