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Insulin signaling pathway components, mammalian uncoordinated-18c (Munc18-c), and forkhead box O1 (FOXO1) are modified with O-GlcNAc
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The review proposes BPC-157 may have its own presence in synaptic vesicles, maintaining vesicle integrity and function while competing for receptor sites and substituting for neurotransmitters when neurons are damaged or depleted
Viral infections can deplete glutathione, driving oxidative stress and mitochondrial dysfunction in Long COVID and ME/CFS
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