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During sepsis, excessive ROS production exacerbates mitochondrial dysfunction, creating a feedback loop that worsens oxidative damage (13,90)

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In a PD mouse model induced by intraperitoneal injection of salsolinol (SAL), and in SAL-treated SH-SY5Y cells, GSH expression was significantly reduced, whereas levels of ROS, Fe 2+ , ACSL4, MDA, 4-HNE, and -Syn were markedly elevated

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