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Niu L-G, Sun N, Liu K-L, Su Q, Qi J, Fu L-Y, et al
The aims of this review are (1) to provide an overview of the clinical and biological overlap between neuropsychiatric long COVID and other fatigue-associated neuropsychiatric disorders, (2) to examine the shared molecular mechanisms of fatigue and depressive symptoms involving acetylcarnitine metabolism, and finally (3) to review the current evidence on acetylcarnitine supplementation studies as a treatment for fatigue-associated syndromes and pharmacological aspects of acetylcarnitine supplementation to provide a conceptual foundation for acetylcarnitine supplementation in neuropsychiatric long COVID syndrome
BPC-157 also appears to modulate the dopamine system
GSH system and ferroptosis in the myocardium Ferroptosis Ferroptosis is a novel form of programmed cell death characterized by the accumulation of iron-catalyzed lipid peroxidation
L-carnitine is a key nutrient in cellular metabolism whose main function is to transport fatty acids into mitochondria to be metabolized and used for cell energy production [1]
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