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rather, it constitutes a metabolically active biochemical processing center that enzymatically biotransforms xenobiotics and endogenous toxins through highly regulated, pathway-specific metabolic processes

In line with this, TXNDC5 K150R mutation remarkably reduced the TXNDC5 ubiquitination, and USP2 knockdown did not increase TXNDC5 ubiquitination (Fig

A Mendelian randomization study from European cohorts confirmed a causal link between TGs, cholesterol levels, and depression phenotypes, indicating that these metabolic factors may bridge the connection between T2DM and depression, potentially mediating their association (256)

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The American journal of clinical nutrition 2024