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Additionally, distinct fluctuations in GSH levels during the oxygen-glucose deprivation model process were monitored with real-time fluorescence imaging
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Glutathione might inhibit the effects of cerebral infarction and enhance antiapoptotic signaling after ischemic stroke, suggesting that GSH may be a potent therapeutic antioxidant that can attenuate severe pathologies after ischemic stroke, and stimulating GSH synthesis through administration of GSH precursors and micronutrients like selenium can optimize GSH and GSH peroxidase for optimal antioxidant defense in cerebral ischemia (155, 156)
The compound has been extensively studied in preclinical research models for its cytoprotective, angiogenic, and tissue-modulating properties across a wide range of biological systems
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