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Here we show that oral administration of the non-calorie sweetener, rare sugar d-allulose (d-psicose), induces GLP-1 release, activates vagal afferent signaling, reduces food intake and promotes glucose tolerance in healthy and obese-diabetic animal models
Mechanistically, we show that, upon GSH depletion, the liver tissue induces the expression of target genes associated with the antioxidant transcription factor NRF2
Commonalities regarding the mechanism of GLP-1 and insulin secretions, such as the sharing of glucose-sensing machinery, including Gs activation and glucose stimulation 32,33 , have been reported 6,34
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