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In animal models, GLP1 RAs like liraglutide and exenatide have been linked to lower voluntary alcohol intake
On the basis of these findings, the authors suggested that the principal mechanism of uncoupling is the oxidation of the zincthiolate center and subsequent release of zinc rather than the BH 4 oxidation process
They are collectively classified as proteopathies, which involve severe disruption in cellular protein homeostasis (proteostasis) associated with protein misfolding as well as disruptions in protein processing and localization (Chowhan et al., 2015)
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