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When medications are either not effective or poorly tolerated, RAI is employed, which commonly renders the patient hypothyroid, thus requiring lifelong supplementation of thyroid hormones

While existing research has confirmed that dysregulated expression of copper transporters (such as ATP7A, CTR1) is a core mechanism inducing cellular cuproptosis in models of endothelial injury induced by disturbed blood flow ( Drawing upon the known property of exosomes to targetedly deliver bioactive molecules, we raise a speculative hypothesis: exosomes released by damaged endothelium might have the theoretical capacity to induce secondary cuproptosis responses in the vascular microenvironment, potentially by encapsulating abnormal copper transporters or cytotoxic lipoylated protein aggregates

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